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71] Campieri M, Ferguson A, Doe W, Persson T, Nilsson L. Oral budesonide is as effective as oral prednisolone in active Crohn’s disease. Gut 1997;41:209–14. [72] Kane SV, Schoenfeld P, Sandborn WJ, Tremaine W, Hofer T, Feagan BG. Systematic review: the effectiveness of budesonide therapy for Crohn’s disease. Aliment Pharmacol Ther 2002;16:1509–17. [73] Lofberg R, Rutgeerts P, Malchow H, Lamers C, Danielsson A, Olaison G, et al. Budesonide prolongs time to relapse in ileal and ileocecal Crohn’s disease: a placebo controlled one year study.

A controlled study of bone mineral density in patients with inflammatory bowel disease. Gut 1995;37:71–6. [102] Cino M, Greenberg GR. Bone mineral density in Crohn’s disease: a longitudinal study of budesonide, prednisone, and nonsteroid therapy. Am J Gastroenterol 2002;97:915–21. A. Katz / Gastroenterol Clin N Am 33 (2004) 171–189 189 [103] Loftus EV, Crowson CS, Sandborn WJ, Tremaine WJ, O’Fallon WM, Melton LJ. Longterm fracture risk in Crohn’s disease: a population-based study in Olmstead County, Minnesota.

Recent insight into the structure and function of the glucocorticoid receptor. J Lab Clin Med 1989;113:404–12. [13] Schleimer RP. Effects of glucocorticoids on inflammatory cells relevant to their therapeutic applications in asthma. Am Respir Dis 1990;141:S59–69. [14] Gillis S, Crabtree GR, Smith KA. Glucocorticoid-induced inhibition of T-cell growth factor production. I. The effect of mitogen-induced lymphocyte proliferation. J Immunol 1979;123:1624–31. [15] Ross JS, Bacon KB, Camp RD. Potent and selective inhibition of in vitro lymphocyte migration by cyclosporine and dexamethasone.

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